Younger For Longer

During aging, mitochondria – the power plants of our cells – become dysfunctional.

Senescent cells are former healthy cells that stopped dividing and secrete substances that damage healthy surrounding cells.

Over time, more and more proteins accumulate inside and outside our cells, interfering with their proper functioning.

When we get older, our cells become increasingly exposed to a hostile environment, characterized by inflammation, senescent cells, dysfunctional stem cells, and deleterious substances.

During aging, our DNA becomes damaged, and telomeres become shorter and dysfunctional, especially in stem cells and fast-dividing cells.

During aging, the epigenome – the molecular machinery that regulates our genes’ activity – becomes disorganized.

As we age, the ends of DNA, known as telomeres, get shorter and cannot protect DNA any longer.

During aging, our cells become less tuned to nutrient signals, which disrupts a cell’s ability to produce energy.

With age, stem cells become dysfunctional or die off, leading to our tissues being less replenished & maintained.

As we grow older, sugar-derived bonds, or crosslinks, are formed between the proteins that make up our tissues, making tissues more stiff.